Apoptosis；MAPK signaling pathway；Acute myeloid leukemia；ErbB signaling pathway；Chemokine signaling pathway；Fc gamma R-mediated phagocytosis；Insulin signaling pathway；Pathways in cancer；Melanoma；Dopaminergic synapse；Colorectal cancer；Estrogen signaling pathway；HTLV-I infection；Toll-like receptor signaling pathway；Glioma；Adipocytokine signaling pathway；HIF-1 signaling pathway；Jak-STAT signaling pathway；Proteoglycans in cancer；Toxoplasmosis；B cell receptor signaling pathway；Small cell lung cancer；Progesterone-mediated oocyte maturation；Non-small cell lung cancer；Measles；Fc epsilon RI signaling pathway；mTOR signaling pathway；PI3K-Akt signaling pathway；Chagas disease (American trypanosomiasis)；Hepatitis C；VEGF signaling pathway；Prostate cancer；Renal cell carcinoma；Tuberculosis；Influenza A；T cell receptor signaling pathway；Osteoclast differentiation；Carbohydrate digestion and absorption； Endometrial cancer；Pancreatic cancer；Chronic myeloid leukemia；Cholinergic synapse；Hepatitis B；Focal adhesion；Epstein-Barr virus infection； Neurotrophin signaling pathway；Tight junction

Moreover, SNHG6-003 functioned as a competitive endogenous RNA (ceRNA), effectively becoming sponge for miR-26a/b and thereby modulating the expression of transforming growth factor-β-activated kinase 1 (TAK1).

The long non-coding RNA, SNHG6-003, functions as a competing endogenous RNA to promote the progression of hepatocellular carcinoma