Hepatitis B；Cell cycle；p53 signaling pathway；PI3K-Akt signaling pathway；Measles；Pathways in cancer； Small cell lung cancer；Non-small cell lung cancer；Viral carcinogenesis；Pancreatic cancer；Glioma；Melanoma；Chronic myeloid leukemia

Furthermore, NEAT1 regulated CDK6 expression in LSCC cells which was mediated by miR-107.

Long noncoding RNA NEAT1 promotes laryngeal squamous cell cancer through regulating miR-107/CDK6 pathway

Hepatitis B；Cell cycle；p53 signaling pathway；PI3K-Akt signaling pathway；Measles；Pathways in cancer； Small cell lung cancer；Non-small cell lung cancer；Viral carcinogenesis；Pancreatic cancer；Glioma；Melanoma；Chronic myeloid leukemia

TUSC7 overexpression increased the expression level of CDK6, which is a downstream target of miR-211-3p, in both RNA and protein level. Furthermore, luciferase reporter assay indicated that TUSC7 could sponge miR-211-3p.

The Novel Long Noncoding RNA TUSC7 Inhibits Proliferation by Sponging MiR-211 in Colorectal Cancer

PI3K-Akt signaling pathway；Cell cycle；Small cell lung cancer；p53 signaling pathway；Hepatitis B； Measles；Pathways in cancer；Viral carcinogenesis；Non-small cell lung cancer；Pancreatic cancer；Glioma；Melanoma；Chronic myeloid leukemia

Taken together, we conclude that circRNA_100290 may function as a competing endogenous RNA to regulate CDK6 expression through sponging up miR-29b family members.

circRNA_100290 plays a role in oral cancer by functioning as a sponge of the miR-29 family.