Hepatitis B；Chemokine signaling pathway；HIF-1 signaling pathway；Jak-STAT signaling pathway；Adipocytokine signaling pathway； Toxoplasmosis；Proteoglycans in cancer；Hepatitis C； Measles；Epstein-Barr virus infection；Pathways in cancer；Acute myeloid leukemia；Pancreatic cancer；Viral carcinogenesis

Moreover, MIAT enhanced STAT3 expression through sequestering miR-181b as a molecular sponge. Furthermore, MiR-181b hindered cell growth, induced cell cycle arrest and promoted apoptosis by directly targeting STAT3.

MIAT promotes proliferation and hinders apoptosis by modulating miR-181b/STAT3 axis in ox-LDL-induced atherosclerosis cell models.

Hepatitis B；Chemokine signaling pathway；HIF-1 signaling pathway；Jak-STAT signaling pathway；Adipocytokine signaling pathway； Toxoplasmosis；Proteoglycans in cancer；Hepatitis C； Measles；Epstein-Barr virus infection；Pathways in cancer；Acute myeloid leukemia；Pancreatic cancer；Viral carcinogenesis

Further analysis revealed that STAT3 and ATG12 are targets of miR-454-3p, initiate HOTAIR deficiency-induced apoptosis and reduce autophagy.

Knockdown of long non-coding RNA HOTAIR increases miR-454-3p by targeting Stat3 and Atg12 to inhibit chondrosarcoma growth

Hepatitis B；Chemokine signaling pathway；HIF-1 signaling pathway；Jak-STAT signaling pathway；Adipocytokine signaling pathway； Toxoplasmosis；Proteoglycans in cancer；Hepatitis C； Measles；Epstein-Barr virus infection；Pathways in cancer；Acute myeloid leukemia；Pancreatic cancer；Viral carcinogenesis

Circ_0005015 acted as miR-519d-3p sponge to inhibit miR-519d-3p activity, leading to increased MMP-2, XIAP, and STAT3 expression.

Identification and Characterization of Circular RNAs as a New Class of Putative Biomarkers in Diabetes Retinopathy