Hepatitis B；Chemokine signaling pathway；HIF-1 signaling pathway；Jak-STAT signaling pathway；Adipocytokine signaling pathway； Toxoplasmosis；Proteoglycans in cancer；Hepatitis C； Measles；Epstein-Barr virus infection；Pathways in cancer；Acute myeloid leukemia；Pancreatic cancer；Viral carcinogenesis

Moreover, MIAT enhanced STAT3 expression through sequestering miR-181b as a molecular sponge. Furthermore, MiR-181b hindered cell growth, induced cell cycle arrest and promoted apoptosis by directly targeting STAT3.

MIAT promotes proliferation and hinders apoptosis by modulating miR-181b/STAT3 axis in ox-LDL-induced atherosclerosis cell models.